The Important Role of Lipoprotein(a) in Cardiovascular Disease: Present and Future Therapeutic Options

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Question 1 of 7

1. Question
Pre-Activity

Currently, how confident are you in your ability to:
1 = not confident, 2 = somewhat confident, 3 = confident, 4 = very confident, 5 = extremely confident, N/A = not applicable to me
- Develop personalized treatment plans for patients with cardiovascular disease
  1 2 3 4 5 N/A
  
  Apply newer scientific advances and therapeutic approaches involving lipoprotein(a) to patients for the treatment of cardiovascular disease
  1 2 3 4 5 N/A
Question 2 of 7

2. Question
Indicate how often you CURRENTLY use each of the following clinical practice strategies for your patients with cardiovascular disease.
*1 = Never, 2 = Not often, 3 = Sometimes, 4 = Often, 5 = Always, N/A = Not applicable to me
- Analyze the science of Lp(a) and its role in the management and prevention of cardiovascular risk
  1 2 3 4 5 N/A
  
  Implement clinical trial data and mechanisms of action of emerging therapeutics to reduce Lp(a)
  1 2 3 4 5 N/A
  
  Incorporate new and emerging scientific data into individualized treatment strategies to optimally manage Lp(a) and improve cardiovascular outcomes
  1 2 3 4 5 N/A
Question 3 of 7

3. Question
1. High lipoprotein(a) is causally related to the increased risk of:
- Aortic stenosis
- Myocardial infarction
- Familial hypercholesterolemia
- All of the above
Question 4 of 7

4. Question
2. Which of the following is true?
- Apolipoprotein(a) kringle number determines Lp(a) isoform size
- Proinflammatory oxidized phospholipids can be carried by apo(a)
- Apolipoprotein(a) closely resembles fibrinogen
- Both a and b
- All of the above
Question 5 of 7

5. Question
3. Regarding Lp(a), which of the following answer choice is correct?
- High Lp(a) is a rare genetic dyslipidemia
- High Lp(a) is associated with a very small increase in risk of ASCVD
- High Lp(a) has been linked with premature MI, childhood stroke and progression of aortic stenosis
- Screening for Lp(a) should not be performed since it does not change management
- Lowering Lp(a) to normal levels would be expected to reduce 1 in 100 MIs in the population
Question 6 of 7

6. Question
4. Which of the following answer choices is true regarding treatment for elevated Lp(a) levels?
- Statin therapy lowers Lp(a) levels by 30% and decreases oxidized phospholipids by a similar amount
- Niacin lowers Lp(a) levels and CVD events by at least 30%
- The reduction of Lp(a) with PCSK9 inhibitors is strongly correlated with the magnitude of LDL-C lowering
- Lipid-apheresis improves symptoms in patients with refractory angina and an elevated Lp(a)
- High fat and low vegetable diets increase Lp(a) levels
Question 7 of 7

7. Question
5. Based on current knowledge, the most effective method to reduce plasma levels of Lp(a) is:
- Inhibit apoB synthesis
- Inhibit apo(a) synthesis
- Inhibit plasminogen synthesis
- Upregulate LDL receptor expression

The Important Role of Lipoprotein(a) in Cardiovascular Disease: Present and Future Therapeutic Options

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